Kev lag luam kws tshuaj
Amino acid muaj ntau yam tshuaj tiv thaiv tus yam ntxwv, uas tuaj yeem siv tau los ua kev siv tes ua ntau yam thiab kev sib txuas ntawm tes-based compounds.Nws yog ib tug ntuj txhais tes uas muaj nyob rau hauv qeb tseem ceeb tshaj plaws ntawm kev sib deev.Amino acids nyob rau hauv cov tshuaj synthesis feem ntau yog hais txog cov tshuaj molecules rau cov qauv qauv ntawm cov tshuaj molecule, pib kev sib deev, los yog synthesis ntawm tes tshuaj raws li ib tug neeg tsis taus.
Anti-hypertension
Kev kub siab tau dhau los ua qhov tshwm sim ntau tshaj plaws ntawm cov kab mob plawv hauv tib neeg cov kab mob plawv, thiab nws tseem yog qhov tseem ceeb tshaj plaws rau cov kab mob plawv thiab cov kab mob cerebrovascular.
Tshuaj tua kab mob
Carbon moldyne tshuaj tua kab mob yog cov tshuaj atypical β -lactam cov tshuaj tua kab mob nrog cov tshuaj tua kab mob tshaj plaws thiab cov tshuaj tua kab mob muaj zog tshaj plaws, vim nws muaj t.nws cov yam ntxwv ntawm β-lactamase stability thiab toxicity tsawg tau dhau los ua cov tshuaj tua kab mob tseem ceeb tshaj plaws rau kev kho cov kab mob loj-.
Anti-HIV
HIV protease inhibitors yog ib qho ntawm peb pawg ntawm cov tshuaj tiv thaiv kab mob HIV, uas tuaj yeem cuam tshuam tus kab mob rov ua dua tshiab thiab dhau los ua AIDS m.yam tshuaj.
Antitumor
Classic dihydrocarbonic acid txo qis enzyme inhibitors (aminotate, aminotrexate, Edutantis, Permeusceri) thiab thymotide.Synthetic enzymes inhibit Leartisa uas muaj L-glutamic acid residue qauv.Lub luag haujlwm tseem ceeb yog txhawm rau txhawm rau ua kom cov polymerization ntawm polymerization ntawm glutamate synthetase hauv lub cev.
Nyob rau hauv daim ntawv ntawm polymerization, daim ntawv ntawm polymerization ntawm glutamic acid tsub kom lub sij hawm khaws cia ntawm cov tshuaj molecules nyob rau hauv lub cell, yog li txhim kho nws.inhibitory nyhuv ntawm hydrogen folic acid txo enzyme.L-glutamic acid yog ib qho tseem ceeb raw khoom rau hluavtaws xws li tshuaj.
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